The effect of inflammation-related lifestyle exposures and interactions with gene variants on long interspersed nuclear element-1 DNA methylation

Epigenomics. 2018 Jun;10(6):785-796. doi: 10.2217/epi-2017-0164. Epub 2018 Jun 11.

Abstract

Aim: To examine the relationship between inflammation-related lifestyle factors and long interspersed nuclear element-1 (LINE-1) DNA methylation, and test for interaction by gene variants involved in one-carbon metabolism.

Patients & methods: The study population consisted of 280 individuals undergoing colonoscopy screening. Multivariable linear regression was employed to examine associations of physical activity, BMI and NSAID use with LINE-1 DNA methylation and interactions with MTR and MTHFR gene variants.

Results: The highest quartile of physical activity compared with the lowest was associated with higher LINE-1 DNA methylation (p = 0.005). Long-term NSAID use and a normal BMI were associated with increased LINE-1 DNA methylation among individuals with the variant MTR allele (p = 0.02; p = 0.03).

Conclusion: This study provides evidence that inflammation-related exposures may influence LINE-1 DNA methylation.

Keywords: BMI; DNA methylation; LINE-1; NSAID use; gene variants; inflammation; lifestyle factors; one-carbon metabolism; physical activity.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • 5-Methyltetrahydrofolate-Homocysteine S-Methyltransferase / genetics
  • Adult
  • Anti-Inflammatory Agents, Non-Steroidal / therapeutic use
  • Body Mass Index
  • DNA Methylation*
  • Exercise
  • Female
  • Genetic Variation
  • Humans
  • Inflammation / genetics*
  • Leukocytes / metabolism*
  • Life Style*
  • Long Interspersed Nucleotide Elements / genetics*
  • Male
  • Methylenetetrahydrofolate Reductase (NADPH2) / genetics
  • Middle Aged

Substances

  • Anti-Inflammatory Agents, Non-Steroidal
  • MTHFR protein, human
  • Methylenetetrahydrofolate Reductase (NADPH2)
  • 5-Methyltetrahydrofolate-Homocysteine S-Methyltransferase
  • MTR protein, human

Grants and funding