Hyperoside and let-7a-5p synergistically inhibits lung cancer cell proliferation via inducing G1/S phase arrest

Gene. 2018 Dec 30:679:232-240. doi: 10.1016/j.gene.2018.09.011. Epub 2018 Sep 7.

Abstract

Lung cancer remains one of the most aggressive human malignancies with a low survival rate. Hyperoside (quercetin-3-O-β-d-galactopyranoside) is a flavonol glycoside with an anti-cancer activity. The microRNA-let-7 was widely regarded as a tumor suppressor in human tumors. Here, we investigated the role of hyperoside and let-7a-5p on the lung cancer cell proliferation, cell cycle and apoptosis in A549 cells in vitro. Our results showed that hyperoside could inhibit the proliferation of A549 cells through inducing apoptosis and G1/S phase arrest. Let-7a-5p could inhibit the proliferation of A549 cells via inhibiting the process of G1/S phase. Additionally, hyperoside and let-7a-5p had a synergetic effect on suppressing the proliferation of A549 cells; microRNA-let-7a-5p directly regulated the expression of CCND1 in A549 cells. Our study illustrated that hyperoside and microRNA-let7a-5p might provide a synergistic effect on anti-cancer, which may provide a new idea for lung cancer treatment.

Keywords: CCND1; Hyperoside; Let-7a-5p; Lung cancer; Proliferation.

MeSH terms

  • 3' Untranslated Regions
  • A549 Cells
  • Cell Proliferation / drug effects
  • Cell Survival / drug effects
  • Cyclin D1 / genetics*
  • G1 Phase Cell Cycle Checkpoints / drug effects
  • Gene Expression Regulation, Neoplastic / drug effects
  • Humans
  • Lung Neoplasms / drug therapy
  • Lung Neoplasms / genetics*
  • MicroRNAs / genetics*
  • Quercetin / analogs & derivatives*
  • Quercetin / pharmacology

Substances

  • 3' Untranslated Regions
  • CCND1 protein, human
  • MicroRNAs
  • mirnlet7 microRNA, human
  • Cyclin D1
  • hyperoside
  • Quercetin