Whole exome sequencing identifies both nuclear and mitochondrial variations in an Iranian family with non-syndromic hearing loss

Mitochondrion. 2019 May:46:321-325. doi: 10.1016/j.mito.2018.08.006. Epub 2018 Sep 8.

Abstract

Genetic contributing factors to non-syndromic hearing loss (NSHL) are remarkably diverse spanning over autosomal to X-linked to mitochondrial inheritance patterns. Facing a quite unconventional pedigree, here we report implementation of whole exome sequencing (WES) to uncover mitochondrial pathogenic variant in a six-generation Iranian family with four cases affected with hereditary NSHL of variable severity. As a result, heteroplasmic transition of A to G at position 1555 of MT-RNR1 gene was identified in all affected individuals co-existing with nuclear c.28G > T (p.A10S) variant in the TRMU gene, only in some patients. The reliability of WES to infer nuclear as well as mitochondrial variants in hearing loss were discussed.

Keywords: 12s rRNA; GJB2 gene; GTPBP3 gene; Non-consanguineous; TFB1M gene; TRMU gene.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Exome Sequencing
  • Female
  • Genetic Diseases, Inborn / genetics*
  • Genetic Diseases, Inborn / pathology
  • Hearing Loss / genetics*
  • Hearing Loss / pathology
  • Humans
  • Iran
  • Mitochondrial Proteins
  • Pedigree
  • Point Mutation*
  • RNA, Mitochondrial / genetics*
  • RNA, Ribosomal / genetics*
  • tRNA Methyltransferases / genetics*

Substances

  • Mitochondrial Proteins
  • RNA, Mitochondrial
  • RNA, Ribosomal
  • RNA, ribosomal, 12S
  • tRNA Methyltransferases
  • TRMU protein, human