MiR-451a suppressed cell migration and invasion in non-small cell lung cancer through targeting ATF2

Eur Rev Med Pharmacol Sci. 2018 Sep;22(17):5554-5561. doi: 10.26355/eurrev_201809_15818.

Abstract

Objective: To explore the role of miR-451a in the migration and invasion of non-small cell lung cancer (NSCLC) cells.

Materials and methods: Quantitative Real time-polymerase chain reaction (qRT-PCR) and Western blot were performed to detect the levels of miR-451a and activating transcription factor 2 (ATF2) in NSCLC. Transwell assay was employed to analyze the migratory and invasive abilities in NSCLC cells. Dual-luciferase reporter assay was applied to confirm the binding condition of miR-451 and its target gene in NSCLC cells.

Results: MiR-451a was downregulated in NSCLC tissues and lung cancer cell lines A549 and NCI-H460, while ATF2 was upregulated. The mRNA level of miR-451a was negatively correlated to ATF2. Additionally, miR-451a regulated cell migration and invasion through targeting ATF2. Furthermore, ATF2 could reverse the inhibitory migration and invasion of A549 cells induced by miR-451a.

Conclusions: MiR-451a inhibits the migratory and invasive abilities of NSCLC cells through ATF2 regulation. The newly identified miR-451a/ATF2 axis provides a novel insight into the pathogenesis ofNSCLC.

MeSH terms

  • A549 Cells
  • Activating Transcription Factor 2 / biosynthesis*
  • Activating Transcription Factor 2 / genetics
  • Carcinoma, Non-Small-Cell Lung / genetics
  • Carcinoma, Non-Small-Cell Lung / metabolism*
  • Carcinoma, Non-Small-Cell Lung / pathology
  • Cell Movement / physiology
  • Humans
  • Lung Neoplasms / genetics
  • Lung Neoplasms / metabolism*
  • Lung Neoplasms / pathology
  • MicroRNAs / biosynthesis*
  • MicroRNAs / genetics
  • Neoplasm Invasiveness / genetics
  • Neoplasm Invasiveness / pathology

Substances

  • ATF2 protein, human
  • Activating Transcription Factor 2
  • MIRN451 microRNA, human
  • MicroRNAs