Lost in Translation: Deciphering the Mechanism of Action of Anti-human CTLA-4

Clin Cancer Res. 2019 Feb 15;25(4):1130-1132. doi: 10.1158/1078-0432.CCR-18-2509. Epub 2018 Oct 5.

Abstract

Despite a number of preclinical studies demonstrating that the activity of anti-CTLA-4 antibodies in murine models of cancer relies on effector T-cell activation and regulatory T cell depletion, the activity of the clinical antibodies remains controversial. To decipher such mechanisms is critical to the development of novel and more potent immunotherapies.See related article by Sharma et al., p. 1233.

Publication types

  • Research Support, Non-U.S. Gov't
  • Comment

MeSH terms

  • Animals
  • CTLA-4 Antigen
  • Forkhead Transcription Factors
  • Humans
  • Immunotherapy
  • Mice
  • Neoplasms*
  • T-Lymphocytes, Regulatory*

Substances

  • CTLA-4 Antigen
  • FOXP3 protein, human
  • Forkhead Transcription Factors
  • Foxp3 protein, mouse