Santin inhibits influenza A virus replication through regulating MAPKs and NF-κB pathways

J Asian Nat Prod Res. 2019 Dec;21(12):1205-1214. doi: 10.1080/10286020.2018.1520221. Epub 2018 Nov 10.

Abstract

Influenza A virus (IAV) causes high morbidity and significant mortality worldwide. Given the limitations of existing vaccination and antiviral drugs, it is urgent to develop new anti-influenza drugs. Flavonoids are natural polyphenolic compounds with broad applications to treatments for influenza infection. In this study, we demonstrated that santin, a flavonoid compound, showed anti-influenza activity in MDCK and THP-1 cells. Mechanistic studies revealed that santin depressed the phosphorylation of p38 MAPK, JNK/SAPK, ERK, and NF-κB factor and subsequently attenuated the expression of inflammatory cytokines in IAV-infected cells. Thus, santin is a potential candidate for the future development of anti-IAV drugs.

Keywords: MAPKs; NF-κB; Santin; inflammatory cytokines; influenza A virus.

MeSH terms

  • Animals
  • Dogs
  • Flavonoids
  • Influenza A virus*
  • Influenza, Human*
  • Madin Darby Canine Kidney Cells
  • Molecular Structure
  • NF-kappa B
  • Signal Transduction
  • Virus Replication

Substances

  • Flavonoids
  • NF-kappa B
  • santin