Double-negative T cells remarkably promote neuroinflammation after ischemic stroke

Proc Natl Acad Sci U S A. 2019 Mar 19;116(12):5558-5563. doi: 10.1073/pnas.1814394116. Epub 2019 Feb 28.

Abstract

CD3+CD4-CD8- T cells (double-negative T cells; DNTs) have diverse functions in peripheral immune-related diseases by regulating immunological and inflammatory homeostasis. However, the functions of DNTs in the central nervous system remain unknown. Here, we found that the levels of DNTs were dramatically increased in both the brain and peripheral blood of stroke patients and in a mouse model in a time-dependent manner. The infiltrating DNTs enhanced cerebral immune and inflammatory responses and exacerbated ischemic brain injury by modulating the FasL/PTPN2/TNF-α signaling pathway. Blockade of this pathway limited DNT-mediated neuroinflammation and improved the outcomes of stroke. Our results identified a critical function of DNTs in the ischemic brain, suggesting that this unique population serves as an attractive target for the treatment of ischemic stroke.

Keywords: DNTs; FasL; T cell-mediated immunity; ischemic stroke; neuroinflammation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aged, 80 and over
  • Animals
  • Brain / immunology
  • Brain Ischemia / immunology*
  • CD3 Complex / immunology*
  • CD4 Antigens / immunology
  • CD8 Antigens / immunology
  • Disease Models, Animal
  • Female
  • Humans
  • Inflammation / immunology
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Microglia / immunology
  • Stroke / immunology*
  • T-Lymphocyte Subsets / immunology*
  • Tumor Necrosis Factor-alpha / metabolism

Substances

  • CD3 Complex
  • CD4 Antigens
  • CD8 Antigens
  • Tumor Necrosis Factor-alpha