T-cell and B-cell perturbations are similar in ART-naive HIV-1 and HIV-1/2 dually infected patients

AIDS. 2019 Jun 1;33(7):1143-1153. doi: 10.1097/QAD.0000000000002185.

Abstract

Background: HIV-2 may slow progression of a subsequently acquired HIV-1 infection through cross-neutralizing antibodies and polyfunctional CD8 T cells. We hypothesized that HIV-1/2 dually infected patients compared with HIV-1-infected patients had more preserved immune maturation subsets and less immune activation of T and B cells.

Methods: ART-naive patients with HIV-1 (n = 83) or HIV-1/2 dual (n = 27) infections were included in this cross-sectional study at an HIV clinic in Guinea-Bissau. Peripheral blood mononuclear cells (PBMCs) were analyzed by flow cytometry according to T-cell maturation and activation, regulatory T-cell fraction, and B-cell maturation and activation.

Results: HIV-1/2 dually infected patients had lower levels of HIV-1 RNA compared with patients with HIV-1 infection, but the levels of total HIV RNA (HIV-1 and HIV-2) were similar in the two patient groups. T-cell maturation, and proportions of regulatory T cells (FoxP3+) were also similar in the two groups. HIV-1/2 dually infected patients had higher proportions of CD4 and CD8 T cells positive for the activation marker CD38, but there was no difference in other T-cell activation markers (CD28, CTLA-4, PD-1). HIV-1/2 dually infected patients also had higher proportions of IgM-only B cells and plasmablasts.

Conclusion: HIV-1/2 was not associated with less immune perturbations than for HIV-1 infection.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • B-Lymphocytes / immunology*
  • Cross-Sectional Studies
  • Female
  • Flow Cytometry
  • Guinea-Bissau
  • HIV Infections / immunology
  • HIV Infections / virology*
  • HIV-1 / isolation & purification*
  • HIV-2 / isolation & purification*
  • Humans
  • Immunophenotyping
  • Leukocytes, Mononuclear / immunology
  • Lymphocyte Activation
  • Male
  • Middle Aged
  • RNA, Viral / blood*
  • T-Lymphocyte Subsets / immunology*
  • Viral Load

Substances

  • RNA, Viral