[Management of non-small cell lung cancer patients harboring activating mutations and CNS progression]

Rev Mal Respir. 2019 May;36(5):583-590. doi: 10.1016/j.rmr.2019.04.003. Epub 2019 Jun 13.
[Article in French]

Abstract

The central nervous system (CNS), through carcinomatous meningitis or solid brain metastases, is the most common site of recurrence in non-small cell lung cancers (NSCLC) with activating mutations. Our retrospective study describes the population of patients with CNS metastases of NSCLC harboring activating mutation with targeted therapy (EGFR, ALK, BRAF, HER2) in 4 French regional reference hospitals. 60 patients were analyzed. The proposed treatments were heterogeneous and included combinations of chemotherapy, targeted therapy and radiotherapy±associated with topical treatments. Median overall survival following CNS metastasis in these patients was 15.8 months for meningitis carcinoma and 26 months for brain metastases. In patients with brain metastases, the addition of targeted therapy treatment allows a significant improvement in median progression free survival from 5.9 months to 10.6 months (HR 0.48 CI95 [0.24 to 0.97] P=0.035). These patients seem therefore benefit from systemic therapy and particularly targeted therapy with better survival than usual.

Keywords: ALK; Cancer bronchique; Cerebral metastases; EGFR cancer; EGFR lung meningitis targeted therapy; Méningite carcinomateuse; Métastases cérébrales.

Publication types

  • Multicenter Study
  • Observational Study

MeSH terms

  • Adult
  • Aged
  • Aged, 80 and over
  • Anaplastic Lymphoma Kinase / genetics
  • Carcinoma, Non-Small-Cell Lung / genetics
  • Carcinoma, Non-Small-Cell Lung / pathology*
  • Carcinoma, Non-Small-Cell Lung / therapy*
  • Central Nervous System Neoplasms / genetics
  • Central Nervous System Neoplasms / secondary*
  • Central Nervous System Neoplasms / therapy*
  • Disease Progression
  • ErbB Receptors / genetics
  • Female
  • Gain of Function Mutation*
  • Humans
  • Lung Neoplasms / genetics
  • Lung Neoplasms / pathology*
  • Lung Neoplasms / therapy*
  • Male
  • Middle Aged
  • Proto-Oncogene Proteins B-raf / genetics
  • Receptor, ErbB-2 / genetics
  • Retrospective Studies

Substances

  • ALK protein, human
  • Anaplastic Lymphoma Kinase
  • EGFR protein, human
  • ERBB2 protein, human
  • ErbB Receptors
  • Receptor, ErbB-2
  • BRAF protein, human
  • Proto-Oncogene Proteins B-raf