Primary mucoepidermoid carcinoma of the liver with CRTC1-MAML2 fusion: a case report

Diagn Pathol. 2019 Jul 27;14(1):84. doi: 10.1186/s13000-019-0863-8.

Abstract

Background: CRTC1-MAML2 fusion is often detected in low- or intermediate-grade salivary mucoepidermoid carcinoma (MEC), and it is associated with a favorable clinical course. Primary MEC of the liver is an extremely rare, aggressive tumor, and no study has investigated CRTC1-MAML2 fusion.

Case presentation: A 79-year-old Japanese female presented with an approx. 5-cm hepatic mass lesion. We surgically resected the lesion under the clinical diagnosis of intrahepatic cholangiocarcinoma. The histological and immunohistochemical findings were consistent with high-grade MEC, consisting of squamoid, mucin-producing, and intermediate tumor cells. Our RT-PCR analysis revealed the presence of CRTC1-MAML2 fusion. This fusion gene was further confirmed by direct sequencing. The patient is still alive almost 10 years after the surgery.

Conclusion: This is the first case report of primary MEC of the liver with CRTC1-MAML2 fusion, with long survival. The present case has significant implications for the entity of primary MEC of the liver which should be distinguished from adenosquamous carcinoma.

Keywords: Adenosquamous carcinoma; Cholangiocellular carcinoma; Liver; MAML2 fusion; Mucoepidermoid carcinoma.

Publication types

  • Case Reports

MeSH terms

  • Aged
  • Biomarkers, Tumor / genetics
  • Carcinoma, Adenosquamous / diagnosis
  • Carcinoma, Adenosquamous / genetics
  • Carcinoma, Adenosquamous / pathology
  • Carcinoma, Mucoepidermoid / genetics
  • Carcinoma, Mucoepidermoid / metabolism
  • Carcinoma, Mucoepidermoid / pathology*
  • DNA-Binding Proteins / genetics
  • Female
  • Gene Expression Regulation, Neoplastic*
  • Humans
  • Liver / pathology
  • Oncogene Proteins, Fusion / genetics
  • Trans-Activators / genetics*
  • Transcription Factors / genetics*

Substances

  • Biomarkers, Tumor
  • CRTC1 protein, human
  • DNA-Binding Proteins
  • MAML2 protein, human
  • Oncogene Proteins, Fusion
  • Trans-Activators
  • Transcription Factors