Protection with phencyclidine against inactivation of 5-HT2 receptors by sulfhydryl-modifying reagents

Biochem Pharmacol. 1988 Sep 1;37(17):3277-83. doi: 10.1016/0006-2952(88)90639-9.

Abstract

We investigated whether phencyclidine (PCP)-induced head-twitch was antagonized in rats by ritanserin, a selective serotonin2 (5-HT2) receptor antagonist, to confirm the involvement of 5-HT neurons in PCP action and to discover whether PCP could protect the binding sites of [3H]PCP and [3H]ketanserin from the inhibitory effect of protein-modifying reagents which affect sulfhydryl groups. PCP (7.5, 10 and 12.5 mg/kg, i.p.)-induced head-twitch was completely antagonized by ritanserin (1 mg/kg, s.c.). Scatchard plots of specific [3H]PCP and [3H]ketanserin binding showed that sulfhydryl-modifying reagent, N-ethylmaleimide (NEM, 100 microM) caused a significant decrease in Bmax without changing Kd. PCP (10 microM) and ritanserin (1 microM) protected [3H]PCP and [3H]ketanserin binding sites from the decrease in the number induced by NEM (100 microM). 5-HT protected [3H]5-HT binding sites from inactivation by NEM, but PCP and ritanserin did not show any effect. On the basis of the present findings, it is concluded that PCP can interact with 5-HT2 receptors directly or allosterically, and 5-HT2 receptors may locate at PCP binding sites in membranes.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Behavior, Animal / drug effects
  • Ethylmaleimide / antagonists & inhibitors
  • Ketanserin / pharmacology
  • Male
  • Phencyclidine / antagonists & inhibitors
  • Phencyclidine / pharmacology*
  • Piperidines / pharmacology
  • Rats
  • Rats, Inbred F344
  • Receptors, Serotonin / drug effects*
  • Ritanserin
  • Serotonin / pharmacology
  • Sulfhydryl Reagents / antagonists & inhibitors*

Substances

  • Piperidines
  • Receptors, Serotonin
  • Sulfhydryl Reagents
  • Ritanserin
  • Serotonin
  • Ketanserin
  • Phencyclidine
  • Ethylmaleimide