Histone demethylase PHF2 activates CREB and promotes memory consolidation

EMBO Rep. 2019 Sep;20(9):e45907. doi: 10.15252/embr.201845907. Epub 2019 Jul 30.

Abstract

Long-term memory formation is attributed to experience-dependent gene expression. Dynamic changes in histone methylation are essential for the epigenetic regulation of memory consolidation-related genes. Here, we demonstrate that the plant homeodomain finger protein 2 (PHF2) histone demethylase is upregulated in the mouse hippocampus during the experience phase and plays an essential role in memory formation. PHF2 promotes the expression of memory-related genes by epigenetically reinforcing the TrkB-CREB signaling pathway. In behavioral tests, memory formation is enhanced by transgenic overexpression of PHF2 in mice, but is impaired by silencing PHF2 in the hippocampus. Electrophysiological studies reveal that PHF2 elevates field excitatory postsynaptic potential (fEPSP) and NMDA receptor-mediated evoked excitatory postsynaptic current (EPSC) in CA1 pyramidal neurons, suggesting that PHF2 promotes long-term potentiation. This study provides insight into the epigenetic regulation of learning and memory formation, which advances our knowledge to improve memory in patients with degenerative brain diseases.

Keywords: CREB; PHF2; hippocampus; learning and memory; lysine methylation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Computational Biology
  • Epigenesis, Genetic / genetics
  • Hippocampus / metabolism
  • Histone Demethylases / genetics
  • Histone Demethylases / metabolism*
  • Homeodomain Proteins / genetics
  • Homeodomain Proteins / metabolism*
  • Male
  • Mass Spectrometry
  • Maze Learning
  • Memory Consolidation / physiology*
  • Mice
  • Mice, Inbred C57BL
  • Mice, Transgenic

Substances

  • Homeodomain Proteins
  • Histone Demethylases
  • PHF2 protein, mouse

Associated data

  • GEO/GSE129148