Arabidopsis mtHSC70-1 physically interacts with the Cox2 subunit of cytochrome c oxidase

Plant Signal Behav. 2020;15(2):1714189. doi: 10.1080/15592324.2020.1714189. Epub 2020 Jan 14.

Abstract

The 70-kD heat shock proteins (HSP70s or HSC70s) function as molecular chaperones and are involved in diverse cellular processes. We recently demonstrated the roles of mitochondrial HSC70-1 (mtHSC70-1) in the establishment of cytochrome c oxidase (COX)-dependent respiration and redox homeostasis in Arabidopsis thaliana. Defects in COX assembly were observed in the mtHSC70-1 knockout lines. The levels of Cox2 (COX subunit 2) proteins in COX complex were markedly lower in the mutants than in wild-type plants; however, the levels of total Cox2 proteins in the mutants were not obviously different from those in wild-type plants, suggesting that the stability of COX or the availability of Cox2 was impaired in the mtHSC70-1 mutants. Here, we further detected the interaction between mtHSC70-1 and Cox2 proteins through co-immunoprecipitation, pull-down and firefly luciferase complementation imaging assays. The results showed that mtHSC70-1 could directly combine Cox2 in vivo and in vitro, providing supporting evidence for the role of mtHSC70-1 in COX assembly.

Keywords: Arabidopsis; Cox2; cytochrome c oxidase; mtHSC70-1.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Arabidopsis / genetics
  • Arabidopsis / metabolism*
  • Arabidopsis Proteins / genetics
  • Arabidopsis Proteins / metabolism*
  • Cyclooxygenase 2 / genetics
  • Cyclooxygenase 2 / metabolism*
  • Electron Transport Complex IV / genetics
  • Electron Transport Complex IV / metabolism*
  • Gene Expression Regulation, Plant
  • HSP70 Heat-Shock Proteins / genetics
  • HSP70 Heat-Shock Proteins / metabolism
  • Mitochondrial Proteins / genetics
  • Mitochondrial Proteins / metabolism
  • Protein Binding

Substances

  • Arabidopsis Proteins
  • HSP70 Heat-Shock Proteins
  • Mitochondrial Proteins
  • Cyclooxygenase 2
  • Electron Transport Complex IV

Grants and funding

This work was supported by the National Natural Science Foundation of China [grant no. 31870222], the Natural Science Foundation of Hebei Province, China [grant no. C2019205055] and Start-up funding from Hebei Normal University, China [grant no. L042018B01].