Abstract
Erythropoietin (EPO) production in the kidney is regulated by the oxygen-sensing transcription factor HIF-1α, which is degraded under normoxic conditions by HIF-prolyl hydroxylase (HIF-PHD). Inhibition of HIF-PHD by roxadustat leads to increased EPO production, better iron absorption, and amelioration of anemia in chronic kidney disease (CKD).
Copyright © 2020. Published by Elsevier Inc.
MeSH terms
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Anemia / metabolism
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Anemia / therapy*
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Glycine / analogs & derivatives*
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Glycine / therapeutic use
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Humans
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Hypoxia-Inducible Factor 1 / drug effects
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Hypoxia-Inducible Factor 1 / metabolism*
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Isoquinolines / therapeutic use*
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Kidney / metabolism
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Kidney / pathology
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Prolyl Hydroxylases / drug effects
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Prolyl Hydroxylases / metabolism
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Renal Insufficiency, Chronic / drug therapy
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Renal Insufficiency, Chronic / metabolism
Substances
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Hypoxia-Inducible Factor 1
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Isoquinolines
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Prolyl Hydroxylases
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Glycine
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roxadustat