Leuconostoc mesenteroides fermentation produces butyric acid and mediates Ffar2 to regulate blood glucose and insulin in type 1 diabetic mice

Sci Rep. 2020 May 13;10(1):7928. doi: 10.1038/s41598-020-64916-2.

Abstract

Type 1 diabetic patients have lower counts of butyric acid-producing bacteria in the dysbiotic gut microbiome. In this study, we demonstrate that a butyric acid-producing Leuconostoc mesenteroides (L. mesenteroides) EH-1 strain isolated from Mongolian curd cheese can reduce blood glucose and IL-6 in the type 1 diabetic mouse model. L. mesenteroides EH-1 fermentation yielded high concentrations of butyric acid both in vitro and in vivo. Butyric acid or L. mesenteroides EH-1 increased the amounts of insulin in Min6 cell culture and streptozotocin (STZ)-induced diabetic mice. Inhibition or siRNA knockdown of free fatty acid receptor 2 (Ffar2) considerably reduced the anti-diabetic effect of probiotic L. mesenteroides EH-1 or butyric acid by lowering the level of blood glucose. We here demonstrate that Ffar2 mediated the effects of L. mesenteroides EH-1 and butryic acid on regulation of blood glucose and insulin in type 1 diabetic mice.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Blood Glucose / drug effects*
  • Butyric Acid / metabolism*
  • Butyric Acid / pharmacology*
  • Cell Line
  • Diabetes Mellitus, Experimental / drug therapy
  • Diabetes Mellitus, Experimental / etiology
  • Diabetes Mellitus, Experimental / metabolism
  • Diabetes Mellitus, Type 1
  • Disease Models, Animal
  • Fermentation*
  • Food Microbiology
  • Gastrointestinal Microbiome
  • Insulin / blood*
  • Leuconostoc mesenteroides / metabolism*
  • Mice
  • Probiotics / administration & dosage
  • Receptors, G-Protein-Coupled / metabolism*

Substances

  • Blood Glucose
  • Ffar2 protein, mouse
  • Insulin
  • Receptors, G-Protein-Coupled
  • Butyric Acid