'Social distancing' of the neuronal nitric oxide synthase from its adaptor protein causes arrhythmogenic trigger-substrate interactions in long QT syndrome

Cardiovasc Res. 2021 Jan 21;117(2):338-340. doi: 10.1093/cvr/cvaa179.

Authors

Andrew Tieu¹, Fadi G Akar^{2

3}

Affiliations

¹ Cardiovascular Research Center, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
² Department of Internal Medicine, Section of Cardiovascular Medicine, Yale New Haven Hospital, New Haven, CT, USA.
³ Section of Cardiovascular Medicine, Cardiovascular Research Center (Y-CVRC), Yale University, New Haven, CT, USA.

No abstract available

Publication types

Editorial
Research Support, N.I.H., Extramural
Comment

MeSH terms

Adaptor Proteins, Signal Transducing / metabolism
Arrhythmias, Cardiac
Humans
Long QT Syndrome* / etiology
Nitric Oxide Synthase Type I / metabolism
Physical Distancing*

Substances

Adaptor Proteins, Signal Transducing
NOS1AP protein, human
NOS1 protein, human
Nitric Oxide Synthase Type I

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