The impact of granulocyte colony-stimulating factor (G-CSF) on thin endometrium of an animal model with rats

Gynecol Endocrinol. 2021 May;37(5):438-445. doi: 10.1080/09513590.2020.1786508. Epub 2020 Jul 2.

Abstract

Materials and methods: After forming of the thin endometrium by uterine injection of 0.2 ml 96% ethyl alcohol to the rats, five days of subcutaneous injections of 40 μg/kg G-CSF or saline were given. Endometrial thickness, immunohistochemically expression of vascular endothelial growth factor receptor-2 (VEGF-R2), proliferative cell nuclear antigen (PCNA) and fibronectin apoptosis with TUNEL method were compared in specimens among four groups of post-model rats.

Results: Endometrial thickness was significantly improved in thin but not in normal endometrium group with GCSF when compared to saline injection. Stromal and glandular epithelial expression of PCNA and pericapillary VEGF-R2 was significantly increased, and apoptosis was significantly decreased with G-CSF. Although fibronectin was also increased with G-CSF in the thin endometrium, the difference was non-significant. In further, G-CSF decreased apoptotic cells and increased expression of PCNA when compared to saline injection in normal endometrium.

Conclusions: G-CSF improves endometrial thickness, proliferation, angiogenesis and DNA fragmentation in thin endometrium.

Keywords: GCSF; IVF; Thin endometrium; granulocyte colony-stimulating factor; recurrent implantation failure.

MeSH terms

  • Animals
  • Disease Models, Animal
  • Drug Evaluation, Preclinical
  • Embryo Implantation
  • Endometrium / drug effects*
  • Female
  • Granulocyte Colony-Stimulating Factor / pharmacology
  • Granulocyte Colony-Stimulating Factor / therapeutic use*
  • Rats
  • Rats, Sprague-Dawley
  • Uterine Diseases / drug therapy*

Substances

  • Granulocyte Colony-Stimulating Factor