Abstract
Cases of neurofibromatosis type 1 (NF1)-associated medullary thyroid carcinoma (MTC) or C-cell hyperplasia are rarely associated with other endocrine tumors or cases with a multiple endocrine neoplasia type 2. In these patients, mutations were detected in the NF1 gene but no mutations were detected in the RET gene. Although vandetanib has been shown to improve progression-free survival in adults with advanced MTC, data in pediatric patients are limited. Herein, we report the use and outcome of vandetanib in a pediatric MTC case in which NF1 gene and RET proto-oncogen mutation were identified together.
Keywords:
Medullary thyroid carcinoma; NF1 gene; RET proto-oncogene; children; vandetanib.
MeSH terms
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Adolescent
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Antineoplastic Agents / therapeutic use*
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Biomarkers, Tumor / genetics*
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Carcinoma, Neuroendocrine / diagnosis
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Carcinoma, Neuroendocrine / drug therapy*
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Carcinoma, Neuroendocrine / genetics
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Germ-Line Mutation*
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Homozygote
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Humans
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Male
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Neurofibromatosis 1 / diagnosis
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Neurofibromatosis 1 / genetics*
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Neurofibromin 1 / genetics*
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Piperidines / therapeutic use*
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Proto-Oncogene Proteins c-ret / genetics*
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Quinazolines / therapeutic use*
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Thyroid Neoplasms / diagnosis
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Thyroid Neoplasms / drug therapy*
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Thyroid Neoplasms / genetics
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Treatment Outcome
Substances
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Antineoplastic Agents
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Biomarkers, Tumor
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NF1 protein, human
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Neurofibromin 1
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Piperidines
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Quinazolines
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Proto-Oncogene Proteins c-ret
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RET protein, human
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vandetanib
Supplementary concepts
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Thyroid cancer, medullary