Abstract
In mouse myocardial infarction, we combined lineage tracing of cardiac macrophages, mapping their ontogeny, with an analysis of their phenotype and phagocytic functions. While embryo-derived macrophages were most abundant in homeostasis, bone marrow-derived MHC-IIlo macrophages increased in both numbers and phagocytic capacity to clear necrotic cardiomyocytes early after ischemia/perfusion injury.
Keywords:
CCR2; MHC-II; embryo; macrophages; myocardial infarction; phagocytosis.
© 2020 The Authors. European Journal of Immunology published by Wiley-VCH GmbH.
Publication types
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Letter
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Research Support, Non-U.S. Gov't
MeSH terms
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Animals
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CX3C Chemokine Receptor 1 / metabolism
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Disease Models, Animal
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Embryo, Mammalian / cytology
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Embryo, Mammalian / immunology
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Histocompatibility Antigens Class II / metabolism*
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Macrophages / immunology*
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Macrophages / pathology
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Mice
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Mice, Transgenic
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Myocardial Infarction / immunology*
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Myocardial Infarction / pathology
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Myocardium / immunology*
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Myocardium / pathology
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Myocytes, Cardiac / immunology
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Myocytes, Cardiac / pathology
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Phagocytosis / immunology
Substances
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CX3C Chemokine Receptor 1
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Cx3cr1 protein, mouse
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Histocompatibility Antigens Class II