In situ hybridization of nucleus basalis neurons shows increased beta-amyloid mRNA in Alzheimer disease

Proc Natl Acad Sci U S A. 1988 Feb;85(4):1227-31. doi: 10.1073/pnas.85.4.1227.

Abstract

To determine which cells within the brain produce beta-amyloid mRNA and to assess expression of the beta-amyloid gene in Alzheimer disease, we analyzed brain tissue from Alzheimer and control patients by in situ hybridization. Our results demonstrate that beta-amyloid mRNA is produced by neurons in the nucleus basalis of Meynert and cerebral cortex and that nucleus basalis perikarya from Alzheimer patients consistently hybridize more beta-amyloid probe than those from controls. These observations support the hypothesis that increased expression of the beta-amyloid gene plays an important role in the deposition of amyloid in the brains of patients with Alzheimer disease.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Alzheimer Disease / metabolism*
  • Amyloid / biosynthesis*
  • Amyloid beta-Peptides
  • Basal Ganglia / analysis*
  • Cerebral Cortex / analysis
  • Humans
  • Neurons / analysis*
  • Nucleic Acid Hybridization
  • RNA, Messenger / analysis*

Substances

  • Amyloid
  • Amyloid beta-Peptides
  • RNA, Messenger