Genetic factors in anthracycline-induced cardiotoxicity in patients treated for pediatric cancer

Expert Opin Drug Metab Toxicol. 2020 Oct;16(10):865-883. doi: 10.1080/17425255.2020.1807937. Epub 2020 Sep 24.

Abstract

Introduction: Cardiovascular diseases are the main cause of treatment-related morbidity and mortality in childhood cancer patients. Anthracyclines, one of the most common chemotherapeutic agents in treatment regimens, are implicated in chemotherapy-induced cardiotoxicity.

Areas covered: This review describes the pharmacogenomic markers related to anthracycline-induced cardiotoxicity affecting childhood cancer patients. We also included a brief overview of the applicability of reported findings to the well-established PETALE cohort of childhood acute lymphoblastic leukemia survivors of the Sainte-Justine University Health Center (Canada).

Expert opinion: The wide variation in interindividual susceptibility to anthracycline-induced cardiotoxicity, along with a multitude of genetic variants detected through association studies, suggests that genetic contributions could be essential for the design of new individualized preventive approaches.

Keywords: Anthracycline-induced cardiotoxicity; childhood cancer; doxorubicin; genetic association studies; pharmacogenomic markers.

Publication types

  • Systematic Review

MeSH terms

  • Animals
  • Anthracyclines / administration & dosage
  • Anthracyclines / adverse effects*
  • Antineoplastic Agents / administration & dosage
  • Antineoplastic Agents / adverse effects*
  • Cardiotoxicity / etiology*
  • Cardiotoxicity / physiopathology
  • Cardiotoxicity / prevention & control
  • Child
  • Genetic Predisposition to Disease
  • Humans
  • Neoplasms / drug therapy
  • Pharmacogenetics

Substances

  • Anthracyclines
  • Antineoplastic Agents