Abstract
Transcription suppressor Musculin (MSC) is enriched in pro-inflammatory Th17 and IL-22-producing ILC3s. While MSC+/+ mice survived DSS-induced colitis, MSC-/- mice showed elevated pro-inflammatory cytokines with severer pathology, reduced body weight, and earlier death. Reversal of colitis symptoms in MSC-/- mice by IL-22 antagonism suggests the existence of MSC:IL-22 regulatory axis.
Keywords:
IL-22; ILC3; Musculin; Th17; Transcription factor.
© 2021 Wiley-VCH GmbH.
Publication types
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Letter
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Research Support, Non-U.S. Gov't
MeSH terms
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Animals
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Basic Helix-Loop-Helix Transcription Factors / genetics
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Basic Helix-Loop-Helix Transcription Factors / immunology*
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Basic Helix-Loop-Helix Transcription Factors / metabolism
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Colitis / chemically induced
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Colitis / genetics
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Colitis / immunology*
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Cytokines / immunology*
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Cytokines / metabolism
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Dextran Sulfate
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Immunity, Innate / immunology
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Inflammation Mediators / immunology*
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Inflammation Mediators / metabolism
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Interleukin-17 / immunology
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Interleukin-17 / metabolism
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Interleukin-22
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Interleukins / genetics
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Interleukins / immunology*
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Interleukins / metabolism
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Lymphocytes / immunology*
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Lymphocytes / metabolism
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Mice
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Mice, Inbred C57BL
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Mice, Knockout
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Mice, Transgenic
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Th17 Cells / immunology*
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Th17 Cells / metabolism
Substances
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Basic Helix-Loop-Helix Transcription Factors
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Cytokines
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Inflammation Mediators
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Interleukin-17
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Interleukins
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Msc protein, mouse
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Dextran Sulfate