Abstract
p300 and CREB-binding protein (CBP) are essential for a multitude of cellular processes. Dysregulation of p300/CBP histone acetyltransferase activity is linked to a broad spectrum of human diseases including cancers. A novel drug-like spirohydantoin (21) has been discovered as a selective orally bioavailable inhibitor of p300/CBP histone acetyltransferase. Lead compound 21 is more potent than the first-in-class lead A-485 in both enzymatic and cellular assays and lacks the off-target inhibition of dopamine and serotonin transporters, that was observed with A-485.
Keywords:
CBP; Histone acetyltransferase; P300; Spirohydantoin.
Copyright © 2021 Elsevier Ltd. All rights reserved.
Publication types
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Research Support, N.I.H., Extramural
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Research Support, U.S. Gov't, Non-P.H.S.
MeSH terms
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Administration, Oral
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Biological Availability
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CREB-Binding Protein / antagonists & inhibitors*
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CREB-Binding Protein / metabolism
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Dose-Response Relationship, Drug
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Drug Discovery*
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E1A-Associated p300 Protein / antagonists & inhibitors*
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E1A-Associated p300 Protein / metabolism
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Enzyme Inhibitors / administration & dosage
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Enzyme Inhibitors / metabolism
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Enzyme Inhibitors / pharmacology*
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Humans
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Hydantoins / administration & dosage
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Hydantoins / metabolism
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Hydantoins / pharmacology*
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Molecular Structure
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Spiro Compounds / administration & dosage
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Spiro Compounds / metabolism
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Spiro Compounds / pharmacology*
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Structure-Activity Relationship
Substances
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Enzyme Inhibitors
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Hydantoins
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Spiro Compounds
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CREB-Binding Protein
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CREBBP protein, human
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E1A-Associated p300 Protein
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EP300 protein, human