Covid-19 and oral diseases: Crosstalk, synergy or association?

Rev Med Virol. 2021 Nov;31(6):e2226. doi: 10.1002/rmv.2226. Epub 2021 Mar 1.

Abstract

The coronavirus disease 2019 (Covid-19) is a viral infection caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) that clinically affects multiple organs of the human body. Cells in the oral cavity express viral entry receptor angiotensin-converting enzyme 2 that allows viral replication and may cause tissue inflammation and destruction. Recent studies have reported that Covid-19 patients present oral manifestations with multiple clinical aspects. In this review, we aim to summarise main signs and symptoms of Covid-19 in the oral cavity, its possible association with oral diseases, and the plausible underlying mechanisms of hyperinflammation reflecting crosstalk between Covid-19 and oral diseases. Ulcers, blisters, necrotising gingivitis, opportunistic coinfections, salivary gland alterations, white and erythematous plaques and gustatory dysfunction were the most reported clinical oral manifestations in patients with Covid-19. In general, the lesions appear concomitant with the loss of smell and taste. Multiple reports show evidences of necrotic/ulcerative gingiva, oral blisters and hypergrowth of opportunistic oral pathogens. SARS-CoV-2 exhibits tropism for endothelial cells and Covid-19-mediated endotheliitis can not only promote inflammation in oral tissues but can also facilitate virus spread. In addition, elevated levels of proinflammatory mediators in patients with Covid-19 and oral infectious disease can impair tissue homeostasis and cause delayed disease resolution. This suggests potential crosstalk of immune-mediated pathways underlying pathogenesis. Interestingly, few reports suggest recurrent herpetic lesions and higher bacterial growth in Covid-19 subjects, indicating SARS-CoV-2 and oral virus/bacteria interaction. Larger cohort studies comparing SARS-CoV-2 negative and positive subjects will reveal oral manifestation of the virus on oral health and its role in exacerbating oral infection.

Keywords: Covid-19; SARS-CoV-2; cytokines; inflammation; oral diseases.

Publication types

  • Research Support, N.I.H., Extramural
  • Review

MeSH terms

  • Angiotensin-Converting Enzyme 2 / genetics
  • Angiotensin-Converting Enzyme 2 / immunology
  • Anosmia / complications
  • Anosmia / immunology
  • Anosmia / pathology
  • Anosmia / virology
  • COVID-19 / complications*
  • COVID-19 / immunology
  • COVID-19 / pathology
  • COVID-19 / virology
  • Dysgeusia / complications
  • Dysgeusia / immunology
  • Dysgeusia / pathology
  • Dysgeusia / virology
  • Gene Expression
  • Gingivitis, Necrotizing Ulcerative / complications*
  • Gingivitis, Necrotizing Ulcerative / immunology
  • Gingivitis, Necrotizing Ulcerative / pathology
  • Gingivitis, Necrotizing Ulcerative / virology
  • Herpesviridae Infections / complications*
  • Herpesviridae Infections / immunology
  • Herpesviridae Infections / pathology
  • Herpesviridae Infections / virology
  • Humans
  • Mouth / immunology
  • Mouth / pathology
  • Mouth / virology
  • Oral Ulcer / complications*
  • Oral Ulcer / immunology
  • Oral Ulcer / pathology
  • Oral Ulcer / virology
  • Periodontal Diseases / complications*
  • Periodontal Diseases / immunology
  • Periodontal Diseases / pathology
  • Periodontal Diseases / virology
  • SARS-CoV-2 / immunology
  • SARS-CoV-2 / pathogenicity
  • Serine Endopeptidases / genetics
  • Serine Endopeptidases / immunology
  • Sialadenitis / complications*
  • Sialadenitis / immunology
  • Sialadenitis / pathology
  • Sialadenitis / virology
  • Stomatitis, Aphthous / complications*
  • Stomatitis, Aphthous / immunology
  • Stomatitis, Aphthous / pathology
  • Stomatitis, Aphthous / virology
  • Xerostomia / complications*
  • Xerostomia / immunology
  • Xerostomia / pathology
  • Xerostomia / virology

Substances

  • ACE2 protein, human
  • Angiotensin-Converting Enzyme 2
  • Serine Endopeptidases
  • TMPRSS2 protein, human