Review of Published Cases of Syncope and Sudden Death in Patients With Severe Aortic Stenosis Documented by Electrocardiography

Am J Cardiol. 2021 Jun 1:148:124-129. doi: 10.1016/j.amjcard.2021.02.023. Epub 2021 Mar 3.

Abstract

The ECG findings during sudden collapse (syncope or sudden death) in severe aortic stenosis (AS) are not well defined. We conducted a comprehensive review of the literature for ECG data during sudden collapse in patients with AS and provided a case report of our own. There were 37 published cases of syncope or sudden death in patients with severe AS which were documented by ECG. Brady- or ventricular arrhythmias were documented in 34 cases (92%). Bradyarrhythmia (n = 24; 71%) was more common at the time of collapse than ventricular tachyarrhythmia (n = 10; 29%). There was slowing of the sinus rate before bradyarrhythmia in the vast majority of patients with bradyarrhythmia but not in those presenting with ventricular tachyarrhythmia (75% vs 0%; p <0.001). ECG evidence of ischemia (ST-segment depression or elevation) was present in most patients with bradyarrhythmia but not in those with ventricular tachyarrhythmia (75% vs 0%; p = 0.011). In conclusion, our findings suggest that left ventricular baroreceptor activation plays a dominant role in the pathophysiology of sudden collapse in patients with severe AS and suggest that ischemia may play a role as well.

Publication types

  • Case Reports
  • Review

MeSH terms

  • Aged
  • Aortic Valve Stenosis / complications
  • Aortic Valve Stenosis / physiopathology*
  • Bicuspid Aortic Valve Disease / complications
  • Bicuspid Aortic Valve Disease / physiopathology*
  • Bradycardia / etiology
  • Bradycardia / physiopathology*
  • Death, Sudden, Cardiac*
  • Electrocardiography
  • Heart Ventricles
  • Humans
  • Male
  • Myocardial Ischemia / etiology
  • Myocardial Ischemia / physiopathology*
  • Pressoreceptors
  • Severity of Illness Index
  • Syncope / etiology
  • Syncope / physiopathology*
  • Tachycardia, Ventricular / etiology
  • Tachycardia, Ventricular / physiopathology*