Benzene and the genotoxicity of its metabolites. I. Transplacental activity in mouse fetuses and in their dams

Mutat Res. 1988 May;208(1):61-7. doi: 10.1016/0165-7992(88)90022-x.

Abstract

Benzene and some of its metabolites (hydroquinone, phenol, catechol, 1,2,4-benzenetriol, p-benzoquinone, o,o'-biphenol, p,p'-biphenol) have been tested for their capability to induce micronuclei in bone marrow cells of pregnant mice and, transplacentally, in fetal liver cells. Dams are scarcely sensitive to the genotoxic activity of benzene and its metabolites while the latter are able to produce only evident toxic effects. Benzene and hydroquinone transplacentally induce micronuclei in fetal liver cells while all other metabolites show weak or negative genotoxicity, although they produce severe cellular toxicity.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Benzene Derivatives / toxicity*
  • Bone Marrow / drug effects
  • Bone Marrow / pathology*
  • Cell Nucleus / drug effects
  • Erythrocytes / cytology
  • Erythrocytes / drug effects
  • Female
  • Fetus / drug effects
  • Maternal-Fetal Exchange*
  • Mice
  • Mutagens*
  • Pregnancy
  • Structure-Activity Relationship

Substances

  • Benzene Derivatives
  • Mutagens