Interferon-induced nuclear factors that bind a shared promoter element correlate with positive and negative transcriptional control

Genes Dev. 1988 Apr;2(4):383-93. doi: 10.1101/gad.2.4.383.

Abstract

Human alpha- and beta-interferons (IFNs) stimulate rapid but transient increases in transcription from a set of previously quiescent genes. Protein synthesis is not required for initial stimulation, but duration of the response is limited to a few hours by a process requiring synthesis of new proteins. An IFN-stimulated response element (ISRE) was identified 5' to an inducible gene by deletion analysis and point mutagenesis, and sequence comparisons with other promoters defined the consensus element YAGTTTC(A/T)YTTTYCC. Two classes of IFN-inducible nuclear factors were found that bind to the ISRE. The most rapidly induced factor appeared without new protein synthesis, whereas a second factor required active protein synthesis for its appearance and maintenance. The kinetics of appearance and loss of these binding activities correlate with the activation and repression of IFN-stimulated genes. These different IFN-activated or induced factors may bind sequentially to the same essential promoter element to first increase and then repress transcription.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Base Sequence
  • DNA
  • DNA-Binding Proteins / physiology
  • Deoxyribonucleases
  • Gene Expression Regulation*
  • HeLa Cells
  • Humans
  • Interferon Type I / physiology*
  • Models, Genetic
  • Mutation
  • Promoter Regions, Genetic*
  • Regulatory Sequences, Nucleic Acid
  • Transcription Factors / isolation & purification*
  • Transcription Factors / physiology
  • Transcription, Genetic*

Substances

  • DNA-Binding Proteins
  • Interferon Type I
  • Transcription Factors
  • DNA
  • Deoxyribonucleases