Nephrotoxicity of butylated hydroxytoluene in phenobarbital-pretreated male rats

Arch Toxicol. 1988 Apr;61(5):359-65. doi: 10.1007/BF00334616.

Abstract

A single large dose of butylated hydroxytoluene (BHT, 1000 mg/kg) in male Fischer 344 rats produced some renal damage, reduced accumulation of p-aminohippuric acid in renal slices, proteinuria and enzymuria, in addition to hepatic damage. Further, prior administration of phenobarbital (80 mg/kg, i.p., daily for 4 days) in the high-dose BHT-treated male rats produced renal damage accompanied by slight tubular necrosis. The renal damage was confirmed by biochemical and histological changes. These changes were dose dependent, with a maximum at 24 h after BHT administration, but had returned to the normal range by 48 h. Female rats, on the other hand, were less susceptible to BHT-induced renal and hepatic damage than male rats. The results indicate sex differences in BHT-induced renal or hepatic damage.

MeSH terms

  • Animals
  • Butylated Hydroxytoluene / toxicity*
  • In Vitro Techniques
  • Kidney / metabolism
  • Kidney / pathology
  • Kidney Diseases / chemically induced*
  • Kidney Diseases / pathology
  • Kidney Diseases / physiopathology
  • Liver / pathology
  • Male
  • Phenobarbital / pharmacology*
  • Rats
  • Rats, Inbred F344
  • Sex Factors
  • Time Factors
  • p-Aminohippuric Acid / urine

Substances

  • Butylated Hydroxytoluene
  • p-Aminohippuric Acid
  • Phenobarbital