On the causal relationships between hyperinsulinaemia, insulin resistance, obesity and dysglycaemia in type 2 diabetes: Reply to Johnson JD [letter]

Diabetologia. 2021 Oct;64(10):2345-2347. doi: 10.1007/s00125-021-05511-6. Epub 2021 Jul 29.

Authors

Nathalie Esser^{1

2}, Kristina M Utzschneider^{1

2}, Steven E Kahn^{3

4}

Affiliations

¹ Veterans Affairs Puget Sound Health Care System, Seattle, WA, USA.
² Division of Metabolism, Endocrinology and Nutrition, Department of Medicine, University of Washington, Seattle, WA, USA.
³ Veterans Affairs Puget Sound Health Care System, Seattle, WA, USA. [email protected].
⁴ Division of Metabolism, Endocrinology and Nutrition, Department of Medicine, University of Washington, Seattle, WA, USA. [email protected].

PMID: 34324020
DOI: 10.1007/s00125-021-05511-6

No abstract available

Keywords: Beta cell function; Dysglycaemia; Glucose effectiveness; Hyperinsulinaemia; Insulin resistance; Obesity; Type 2 diabetes.

Publication types

Letter
Research Support, N.I.H., Extramural
Research Support, Non-U.S. Gov't
Research Support, U.S. Gov't, Non-P.H.S.
Comment

MeSH terms

Diabetes Mellitus, Type 2* / complications
Humans
Hyperinsulinism*
Insulin Resistance*
Obesity / complications