Mycobacterium tuberculosis Phosphoribosyltransferase Promotes Bacterial Survival in Macrophages by Inducing Histone Hypermethylation in Autophagy-Related Genes

Front Cell Infect Microbiol. 2021 Jul 26:11:676456. doi: 10.3389/fcimb.2021.676456. eCollection 2021.

Abstract

Mycobacterium tuberculosis (Mtb) inhibits autophagy to promote its survival in host cells. However, the molecular mechanisms by which Mtb inhibits autophagy are poorly understood. Here, we report a previously unknown mechanism in which Mtb phosphoribosyltransferase (MtbPRT) inhibits autophagy in an mTOR, negative regulator of autophagy, independent manner by inducing histone hypermethylation (H3K9me2/3) at the Atg5 and Atg7 promoters by activating p38-MAPK- and EHMT2 methyltransferase-dependent signaling pathways. Additionally, we find that MtbPRT induces EZH2 methyltransferase-dependent H3K27me3 hypermethylation and reduces histone acetylation modifications (H3K9ac and H3K27ac) by upregulating histone deacetylase 3 to inhibit autophagy. In summary, this is the first demonstration that Mtb inhibits autophagy by inducing histone hypermethylation in autophagy-related genes to promote intracellular bacterial survival.

Keywords: Autophagy; Histone hypermethylation; MAPK pathway; Mycobacterium tuberculosis; Tuberculosis; epigenetic modification.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Autophagy
  • Histones* / metabolism
  • Macrophages / metabolism
  • Macrophages / microbiology*
  • Methylation
  • Mycobacterium tuberculosis* / enzymology
  • Mycobacterium tuberculosis* / genetics
  • Pentosyltransferases / metabolism*
  • Signal Transduction

Substances

  • Histones
  • Pentosyltransferases