NEK6 is an injury-responsive kinase cooperating with STAT3 in regulation of reactive astrogliosis

Glia. 2022 Feb;70(2):273-286. doi: 10.1002/glia.24104. Epub 2021 Oct 13.

Abstract

In response to brain injury, resident astrocytes become reactive and play dynamic roles in neural repair and regeneration. The signaling pathways underlying such reactive astrogliosis remain largely unclear. We here show that NEK6, a NIMA-related serine/threonine protein kinase, is rapidly induced following pathological stimulations and plays critical roles in reactive astrogliosis. Enhanced NEK6 expression promotes reactive astrogliosis and exacerbates brain lesions; and conversely, NEK6 downregulation dampens injury-induced astrocyte reactivity and reduces lesion size. Mechanistically, NEK6 associates with and phosphorylates STAT3. Kinase activity of NEK6 is required for induction of GFAP and PCNA, markers of reactive astrogliosis. Interestingly, NEK6 is also localized in the nucleus and binds to STAT3-responsive genomic elements in astrocytes. These results indicate that NEK6 constitutes a molecular target for the regulation of reactive astrogliosis.

Keywords: NEK6; STAT3; glial scar; neuroinflammation; reactive astrogliosis; traumatic brain injury.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Astrocytes* / metabolism
  • Brain Injuries
  • Glial Fibrillary Acidic Protein
  • Gliosis* / pathology
  • Humans
  • NIMA-Related Kinases* / genetics
  • NIMA-Related Kinases* / metabolism
  • Phosphorylation
  • Proliferating Cell Nuclear Antigen
  • STAT3 Transcription Factor* / metabolism
  • Signal Transduction

Substances

  • GFAP protein, human
  • Glial Fibrillary Acidic Protein
  • PCNA protein, human
  • Proliferating Cell Nuclear Antigen
  • STAT3 Transcription Factor
  • STAT3 protein, human
  • NEK6 protein, human
  • NIMA-Related Kinases