Investigation of lipid peroxidation in human low density lipoprotein

Free Radic Res Commun. 1987;3(1-5):233-42. doi: 10.3109/10715768709069788.

Abstract

Human plasma low density lipoprotein (LDL) exposed to oxygen saturated buffer becomes depleted of alpha-tocopherol within 3 to 6 hours. Thereafter, lipid peroxidation commences as evidenced by the loss of 18:2 (67 nmol/mg LDL) and 20:4 (12 nmol/mg LDL) and the concomitant formation of 4-hydroxynonenal (0.28 nmol/mg LDL) and fluorescent compounds. The major fluorophor in apo B of oxidized LDL has an excitation maximum at 355 nm and an emission maximum at 430 nm. A fluorophor with the same spectral properties is produced in apo B, if LDL is incubated with 4-hydroxynonenal, whereas malonaldehyde gives a fluorophor with excitation and emission maxima at 400/470 nm. Three-dimensional fluorescence spectroscopy proved to be an useful tool in analysing the complex fluorescence of apo B.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aldehydes / metabolism
  • Apolipoproteins B / blood
  • Buffers
  • Humans
  • Lipid Peroxidation*
  • Lipoproteins, LDL / metabolism*
  • Spectrometry, Fluorescence
  • Vitamin E / metabolism

Substances

  • Aldehydes
  • Apolipoproteins B
  • Buffers
  • Lipoproteins, LDL
  • Vitamin E
  • 4-hydroxy-2-nonenal