Brefeldin A arrests the intracellular transport of a precursor of complement C3 before its conversion site in rat hepatocytes

FEBS Lett. 1987 Apr 6;214(1):135-8. doi: 10.1016/0014-5793(87)80028-5.

Abstract

The effects of brefeldin A on intracellular transport and posttranslational modification of complement C3 (C3) were studied in primary culture of rat hepatocytes. In the control culture C3 was synthesized as a precursor (pro-C3), which was processed to the mature form with alpha- and beta-subunits before its discharge into the medium. In the presence of brefeldin A the secretion of C3 was strongly blocked, resulting in accumulation of pro-C3. However, after a prolonged interval the mature form of C3 was finally secreted. The results indicate that brefeldin A impedes translocation of pro-C3 to the Golgi complex where pro-C3 is converted to the mature form, but not its proteolytic processing, in contrast to the effects of monensin and weakly basic amines.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Biological Transport, Active / drug effects
  • Brefeldin A
  • Cells, Cultured
  • Complement C3 / metabolism*
  • Cyclopentanes / pharmacology*
  • Golgi Apparatus / metabolism
  • Liver / drug effects*
  • Liver / immunology
  • Liver / metabolism
  • Protein Precursors / metabolism*
  • Protein Processing, Post-Translational / drug effects
  • Rats

Substances

  • Complement C3
  • Cyclopentanes
  • Protein Precursors
  • Brefeldin A