Scorpion venom inhibits carbamylcholine-induced 86rubidium efflux from rat parotid acini

FEBS Lett. 1987 Jul 27;219(2):451-4. doi: 10.1016/0014-5793(87)80270-3.

Abstract

The muscarinic agonist carbamylcholine stimulated 5-fold 86Rb efflux from preloaded rat parotid acini. Apamin was without effect on this carbamylcholine-induced 86Rb efflux. By contrast, the venom from Leiurus quinquestriatus (a scorpion from Israel) inhibited non-competitively this efflux while being without effect on the carbamylcholine-stimulated 45Ca efflux and amylase release. This heat-resistant inhibitory effect of the venom was destroyed by boiling in the presence of dithiothreitol. These results suggest that the venom from L. quinquestriatus contains a toxin capable to block apamin-insensitive calcium-activated potassium channels in rat parotid acini.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Carbachol / antagonists & inhibitors
  • Carbachol / pharmacology*
  • Female
  • In Vitro Techniques
  • Kinetics
  • Parotid Gland / cytology
  • Parotid Gland / drug effects
  • Parotid Gland / metabolism*
  • Rats
  • Rats, Inbred Strains
  • Rubidium / metabolism*
  • Scorpion Venoms / pharmacology*

Substances

  • Scorpion Venoms
  • Carbachol
  • Rubidium