The antibiotic W341C, its ion transport properties and inhibitory effects on mitochondrial substrate oxidation

J Antibiot (Tokyo). 1987 Jun;40(6):887-93. doi: 10.7164/antibiotics.40.887.

Abstract

We have examined the ion transport properties and the inhibition of rat liver mitochondrial substrate oxidation by the antibiotic W341C. W341C was able to transport 22Na+ and 42K+ across a bulk carbon tetrachloride layer. A preference was shown for K+ transport. With equal molar antibiotic concentrations, W341C transported 42K+ at a greater rate than the K+-selective ionophore nigericin, but transported 22Na+ at a lesser rate than the Na+-selective ionophore monensin. Like nigericin, W341C was able to deplete mitochondrial K+, but not Mg2+ nor Ca2+. The inhibition of mitochondrial substrate oxidation by W341C paralleled the patterns obtained with nigericin. These data indicate that W341C is a K+-selective ionophore that inhibits mitochondrial substrate oxidation by a mechanism analogous to that of nigericin.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Anti-Bacterial Agents
  • Arsenates / pharmacology
  • Calcium / metabolism
  • Carbon Tetrachloride
  • Cations
  • Ethers, Cyclic / metabolism
  • Ethers, Cyclic / pharmacology
  • Magnesium / metabolism
  • Mitochondria, Liver / drug effects*
  • Mitochondria, Liver / metabolism
  • Monensin / metabolism
  • Nigericin / metabolism
  • Nigericin / pharmacology
  • Oxidation-Reduction
  • Oxygen Consumption / drug effects
  • Potassium / metabolism*
  • Rats
  • Sodium / metabolism*

Substances

  • Anti-Bacterial Agents
  • Arsenates
  • Cations
  • Ethers, Cyclic
  • W 341C
  • Monensin
  • Sodium
  • Carbon Tetrachloride
  • Magnesium
  • arsenic acid
  • Nigericin
  • Potassium
  • Calcium