Nitric oxide-driven modifications of lipoic arm inhibit α-ketoacid dehydrogenases

Nat Chem Biol. 2023 Mar;19(3):265-274. doi: 10.1038/s41589-022-01153-w. Epub 2022 Oct 20.

Abstract

Pyruvate dehydrogenase complex (PDHC) and oxoglutarate dehydrogenase complex (OGDC), which belong to the mitochondrial α-ketoacid dehydrogenase family, play crucial roles in cellular metabolism. These multi-subunit enzyme complexes use lipoic arms covalently attached to their E2 subunits to transfer an acyl group to coenzyme A (CoA). Here, we report a novel mechanism capable of substantially inhibiting PDHC and OGDC: reactive nitrogen species (RNS) can covalently modify the thiols on their lipoic arms, generating a series of adducts that block catalytic activity. S-Nitroso-CoA, a product between RNS and the E2 subunit's natural substrate, CoA, can efficiently deliver these modifications onto the lipoic arm. We found RNS-mediated inhibition of PDHC and OGDC occurs during classical macrophage activation, driving significant rewiring of cellular metabolism over time. This work provides a new mechanistic link between RNS and mitochondrial metabolism with potential relevance for numerous physiological and pathological conditions in which RNS accumulate.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • 3-Methyl-2-Oxobutanoate Dehydrogenase (Lipoamide)
  • Arm*
  • Multienzyme Complexes
  • Nitric Oxide*
  • Pyruvate Dehydrogenase Complex / metabolism

Substances

  • 3-Methyl-2-Oxobutanoate Dehydrogenase (Lipoamide)
  • Nitric Oxide
  • Pyruvate Dehydrogenase Complex
  • Multienzyme Complexes