A possible mechanism of increased sodium influx in red cells with abnormal membrane lipid levels induced by phospholipase A2

Am J Hematol. 1987 Sep;26(1):17-26. doi: 10.1002/ajh.2830260103.

Abstract

Membrane transport--sodium (Na+) influx and calcium (Ca2+) uptake--was examined in human mature red cells treated with phospholipase A2 (PLase A2) from snake venom. PLase A2-induced conversion of phosphatidyl choline (PC) to lysophosphatidyl choline (L-PC) was associated with a marked increase in Na+ influx and Ca2+ uptake. After L-PC was removed from the cell membrane of the PLase A2-treated red cells in the presence of albumin, an additional increase in Ca2+ transport was observed. These results indicate that membrane lipid abnormalities, such as increased L-PC and/or a loss of total lipids, appear to induce increased membrane transport.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenosine Triphosphate / blood
  • Biological Transport
  • Calcium / blood
  • Erythrocyte Membrane / metabolism*
  • Humans
  • In Vitro Techniques
  • Lysophosphatidylcholines / blood
  • Membrane Lipids / blood*
  • Phosphatidylcholines / blood
  • Phospholipases / blood*
  • Phospholipases A / blood*
  • Phospholipases A2
  • Potassium / blood
  • Serum Albumin / physiology
  • Sodium / blood*

Substances

  • Lysophosphatidylcholines
  • Membrane Lipids
  • Phosphatidylcholines
  • Serum Albumin
  • Adenosine Triphosphate
  • Sodium
  • Phospholipases
  • Phospholipases A
  • Phospholipases A2
  • Potassium
  • Calcium