Correction: Targeting the overexpressed mitochondrial protein VDAC1 in a mouse model of Alzheimer's disease protects against mitochondrial dysfunction and mitigates brain pathology

Transl Neurodegener. 2023 Jan 11;12(1):2. doi: 10.1186/s40035-023-00335-3.

Ankit Verma^#^{1

2}, Anna Shteinfer-Kuzmine^#², Nikita Kamenetsky^{1

2}, Srinivas Pittala^{1

2}, Avijit Paul^{1

2}, Edna Nahon Crystal³, Alberto Ouro^{4

5}, Vered Chalifa-Caspi⁶, Swaroop Kumar Pandey², Alon Monsonego⁷, Noga Vardi¹, Shira Knafo^{2

4}, Varda Shoshan-Barmatz^{8

9}

¹ Department of Life Sciences, Ben-Gurion University of the Negev, 84105, Beer-Sheva, Israel.
² National Institute for Biotechnology in the Negev, Ben-Gurion University of the Negev, 84105, Beer-Sheva, Israel.
³ Achva Academic College, 79804, Shikmim, Israel.
⁴ Department of Physiology, Faculty of Health Sciences, Ben-Gurion University of the Negev, 84105, Beer-Sheva, Israel.
⁵ NeuroAging Group (NEURAL), Clinical Neurosciences Research Laboratory (LINC), Health Research Institute of Santiago de Compostela (IDIS), 15706, Santiago de Compostela, Spain.
⁶ Ilse Katz Institute for Nanoscale Science and Technology, Ben-Gurion University of the Negev, 84105, Beer-Sheva, Israel.
⁷ The Shraga Segal Department of Microbiology, Immunology, and Genetics, Faculty of Health Sciences, Ben-Gurion University of the Negev, 84105, Beer-Sheva, Israel.
⁸ Department of Life Sciences, Ben-Gurion University of the Negev, 84105, Beer-Sheva, Israel. [email protected].
⁹ National Institute for Biotechnology in the Negev, Ben-Gurion University of the Negev, 84105, Beer-Sheva, Israel. [email protected].

^# Contributed equally.

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