Anti-Vascular Endothelial Growth Factor Therapy Abolishes Glioma-Associated Endothelial Cell-Induced Tumor Invasion

J Mol Neurosci. 2023 Mar;73(2-3):104-116. doi: 10.1007/s12031-023-02099-x. Epub 2023 Jan 18.

Abstract

Tumor-remodeled endothelial cells not only facilitate the formation of tumor angiogenesis but also promote tumorigenesis. In this study, we aimed to explore the interaction between glioma-associated endothelial cells (GAEs) and glioma cells. We found that different subtypes of glioma owned distinct GAE abundance. Glioma patients with high GAE abundance exhibited poor prognosis. Both the results of the bioinformatics analysis and the in vitro co-culture system assay revealed that GAE promoted glioma cell invasion. Besides, anti-vascular endothelial growth factor (VEGF) therapy partially abolished the effects of GAE on gliomas. Moreover, anti-VEGF therapy upregulated IL-2 expression in GAE, and exogenous IL-2 administration inhibits GAE-induced glioma cell invasion. Collectively, our present study provides a novel outstanding of the interaction between GAE and glioma cells.

Keywords: Anti-VEGF therapy; Glioma; Glioma-associated endothelial cells; Invasion.

MeSH terms

  • Brain Neoplasms* / drug therapy
  • Brain Neoplasms* / pathology
  • Cell Line, Tumor
  • Endothelial Cells / metabolism
  • Endothelial Growth Factors / pharmacology
  • Glioma* / drug therapy
  • Glioma* / metabolism
  • Humans
  • Interleukin-2 / pharmacology
  • Neovascularization, Pathologic / drug therapy
  • Neovascularization, Pathologic / metabolism
  • Vascular Endothelial Growth Factor A / genetics

Substances

  • Endothelial Growth Factors
  • Vascular Endothelial Growth Factor A
  • Interleukin-2