Defect in the split proteins of 30-S ribosomal subunits and under-methylation of 16-S ribosomal RNA in a polyamine-requiring mutant of Escherichia coli grown in the absence of polyamines

Eur J Biochem. 1979 Jan 15;93(2):345-53. doi: 10.1111/j.1432-1033.1979.tb12829.x.

Abstract

Polyphenylalanine synthesis was carried out with Escherichia coli Q13 50-S ribosomal subunits and reconstituted 30-S particles containing different combinations of 23-S core particles and 30-S subunit split proteins obtained from a polyamine-requiring mutant of E. coli during its growth in the presence or absence of putrescine. It was concluded that the defect in the amount of some kinds of 30-S subunit split proteins was responsible for the decrease of polypeptide synthesis in a polyamine-requiring mutant of E. coli grown in the absence of polyamines. The methylation of 16-S RNA during growth in the absence of putrescine was decreased, while the degree of methylation of 23-S RNA did not change significantly. The decrease in methylation of 16-S RNA in the absence of putrescine was due mainly to a decrease of methylation of adenine. The relationship between the decrease of polypeptide synthetic activity of 30-S ribosomal subunits obtained from a polyamine-requiring mutant of E. coli grown in the absence of polyamines and the decrease of methylation of 16-S RNA is discussed.

MeSH terms

  • Aminoglycosides
  • Anti-Bacterial Agents / pharmacology
  • Escherichia coli / drug effects
  • Escherichia coli / metabolism*
  • Magnesium / pharmacology
  • Methylation
  • Mutation
  • N-Formylmethionine
  • Protein Biosynthesis / drug effects
  • Putrescine / pharmacology*
  • RNA, Ribosomal / metabolism*
  • RNA, Transfer / metabolism
  • Ribosomal Proteins / biosynthesis*
  • Ribosomes / drug effects
  • Ribosomes / metabolism*
  • Spermidine / pharmacology*

Substances

  • Aminoglycosides
  • Anti-Bacterial Agents
  • RNA, Ribosomal
  • Ribosomal Proteins
  • N-Formylmethionine
  • RNA, Transfer
  • Magnesium
  • Spermidine
  • Putrescine