COVID-19: Potassium Contributes to Pathologies that Cause Disability
Infect Disord Drug Targets
.
2023;23(7):39-41.
doi: 10.2174/1871526523666230529163610.
Authors
Ziad Fajloun
1
2
,
Yingliang Wu
3
,
Zhijian Cao
3
,
Hervé Kovacic
4
,
Jean-Marc Sabatier
4
Affiliations
1
Department of Biology, Faculty of Sciences, Lebanese University, Campus Michel Slayman Ras Maska, 1352 Tripoli, Lebanon.
2
Laboratory of Applied Biotechnology (LBA3B), Azm Center for Research in Biotechnology and its Applications, EDST, Lebanese.
3
State Key Laboratory of Virology, Modern Virology Research Center, College of Life Sciences, Wuhan University, Wuhan 430072, China.
4
Institute of NeuroPhysiopathology (INP), UMR 7051, Aix-Marseille University, Marseille 13385, France.
PMID:
37581327
DOI:
10.2174/1871526523666230529163610
No abstract available
Keywords:
AT1R receptor; COVID-19; Electrolyte; Hypokalemia; Long COVID; Potassium; RAS; SARS-CoV-2.
Publication types
Editorial
MeSH terms
COVID-19*
Humans
Potassium
Renin-Angiotensin System
SARS-CoV-2
Substances
Potassium