Effect of diltiazem on ischemic myocardial depolarization and extracellular K+ accumulation

Eur J Pharmacol. 1986 Aug 15;127(3):261-5. doi: 10.1016/0014-2999(86)90372-9.

Abstract

Diltiazem retards ischemic arrhythmias and reduces cellular depolarization, as inferred from recordings of T-Q segment depression (delta T-Q). To explore this further, we correlated delta T-Q with the extracellular K+ electrode potential (delta EK) during serial ischemic trials. delta T-Q and delta EK were uniform in control trials, but decreased markedly in trials that immediately followed diltiazem infusion (0.5 mg/kg). delta EK at 2 min of ischemia was reduced from 11.8 +/- 1.3 to 7.4 +/- 1.2 mV; while delta T-Q was reduced from 7.2 +/- 0.5 to 4.4 +/- 0.7 mV. The effect of diltiazem on ischemic depolarization is largely, but not entirely explained by reduction of delta EK.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Anti-Arrhythmia Agents / pharmacology*
  • Coronary Disease / drug therapy
  • Coronary Disease / physiopathology*
  • Diltiazem / pharmacology*
  • Diltiazem / therapeutic use
  • Dogs
  • Extracellular Space / metabolism
  • Heart / drug effects*
  • Membrane Potentials / drug effects
  • Myocardium / metabolism
  • Potassium / metabolism*

Substances

  • Anti-Arrhythmia Agents
  • Diltiazem
  • Potassium