Dental pulp stem cells ameliorate D-galactose-induced cardiac ageing in rats

PeerJ. 2024 May 21:12:e17299. doi: 10.7717/peerj.17299. eCollection 2024.

Abstract

Background: Ageing is a key risk factor for cardiovascular disease and is linked to several alterations in cardiac structure and function, including left ventricular hypertrophy and increased cardiomyocyte volume, as well as a decline in the number of cardiomyocytes and ventricular dysfunction, emphasizing the pathological impacts of cardiomyocyte ageing. Dental pulp stem cells (DPSCs) are promising as a cellular therapeutic source due to their minimally invasive surgical approach and remarkable proliferative ability.

Aim: This study is the first to investigate the outcomes of the systemic transplantation of DPSCs in a D-galactose (D-gal)-induced rat model of cardiac ageing. Methods. Thirty 9-week-old Sprague-Dawley male rats were randomly assigned into three groups: control, ageing (D-gal), and transplanted groups (D-gal + DPSCs). D-gal (300 mg/kg/day) was administered intraperitoneally daily for 8 weeks. The rats in the transplantation group were intravenously injected with DPSCs at a dose of 1 × 106 once every 2 weeks.

Results: The transplanted cells migrated to the heart, differentiated into cardiomyocytes, improved cardiac function, upregulated Sirt1 expression, exerted antioxidative effects, modulated connexin-43 expression, attenuated cardiac histopathological alterations, and had anti-senescent and anti-apoptotic effects.

Conclusion: Our results reveal the beneficial effects of DPSC transplantation in a cardiac ageing rat model, suggesting their potential as a viable cell therapy for ageing hearts.

Keywords: Cardiac aging; D-galactose; Dental pulp stem cell; Rat model.

MeSH terms

  • Aging / physiology
  • Animals
  • Apoptosis / drug effects
  • Cell Differentiation / drug effects
  • Connexin 43 / metabolism
  • Dental Pulp* / cytology
  • Disease Models, Animal
  • Galactose*
  • Male
  • Myocytes, Cardiac* / drug effects
  • Myocytes, Cardiac* / metabolism
  • Myocytes, Cardiac* / transplantation
  • Rats
  • Rats, Sprague-Dawley*
  • Sirtuin 1 / metabolism
  • Stem Cell Transplantation / methods
  • Stem Cells / cytology
  • Stem Cells / metabolism

Substances

  • Galactose
  • Sirtuin 1
  • Connexin 43
  • Sirt1 protein, rat

Grants and funding

This research project was funded by the Deanship of Research and Graduate Studies, Ajman University, UAE (grant no. 2022-IRG-MED-5). The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.