Sex-biased immunogenicity of a mucosal subunit vaccine against SARS-CoV-2 in mice

Front Immunol. 2024 May 21:15:1386243. doi: 10.3389/fimmu.2024.1386243. eCollection 2024.

Abstract

Introduction: Current vaccines against COVID-19 administered via parenteral route have limited ability to induce mucosal immunity. There is a need for an effective mucosal vaccine to combat SARS-CoV-2 virus replication in the respiratory mucosa. Moreover, sex differences are known to affect systemic antibody responses against vaccines. However, their role in mucosal cellular responses against a vaccine remains unclear and is underappreciated.

Methods: We evaluated the mucosal immunogenicity of a booster vaccine regimen that is recombinant protein-based and administered intranasally in mice to explore sex differences in mucosal humoral and cellular responses.

Results: Our results showed that vaccinated mice elicited strong systemic antibody (Ab), nasal, and bronchiole alveolar lavage (BAL) IgA responses, and local T cell immune responses in the lung in a sex-biased manner irrespective of mouse genetic background. Monocytes, alveolar macrophages, and CD103+ resident dendritic cells (DCs) in the lungs are correlated with robust mucosal Ab and T cell responses induced by the mucosal vaccine.

Discussion: Our findings provide novel insights into optimizing next-generation booster vaccines against SARS-CoV-2 by inducing spike-specific lung T cell responses, as well as optimizing mucosal immunity for other respiratory infections, and a rationale for considering sex differences in future vaccine research and vaccination practice.

Keywords: SARS-CoV-2; and sex differences; innate immunity; lung tissue-resident T cells; mucosal vaccine.

MeSH terms

  • Administration, Intranasal
  • Animals
  • Antibodies, Viral* / blood
  • Antibodies, Viral* / immunology
  • COVID-19 Vaccines* / immunology
  • COVID-19* / immunology
  • COVID-19* / prevention & control
  • COVID-19* / virology
  • Dendritic Cells / immunology
  • Female
  • Immunity, Humoral
  • Immunity, Mucosal*
  • Immunization, Secondary
  • Immunogenicity, Vaccine*
  • Immunoglobulin A / immunology
  • Lung / immunology
  • Lung / virology
  • Male
  • Mice
  • Mice, Inbred C57BL
  • SARS-CoV-2* / immunology
  • Sex Factors
  • Spike Glycoprotein, Coronavirus / immunology
  • T-Lymphocytes / immunology
  • Vaccines, Subunit* / administration & dosage
  • Vaccines, Subunit* / immunology

Substances

  • COVID-19 Vaccines
  • Vaccines, Subunit
  • Antibodies, Viral
  • Spike Glycoprotein, Coronavirus
  • Immunoglobulin A
  • spike protein, SARS-CoV-2

Grants and funding

The author(s) declare financial support was received for the research, authorship, and/or publication of this article. This work was supported by funding from the Intramural Research Program, National Institutes of Health, National Cancer Institute, Center for Cancer Research funding Z01 BC-011941 to JAB.