Dipeptidyl peptidases and E3 ligases of N-degron pathways cooperate to regulate protein stability

J Cell Biol. 2024 Aug 5;223(8):e202311035. doi: 10.1083/jcb.202311035. Epub 2024 Jun 14.

Abstract

N-degrons are short sequences located at protein N-terminus that mediate the interaction of E3 ligases (E3s) with substrates to promote their proteolysis. It is well established that N-degrons can be exposed following protease cleavage to allow recognition by E3s. However, our knowledge regarding how proteases and E3s cooperate in protein quality control mechanisms remains minimal. Using a systematic approach to monitor the protein stability of an N-terminome library, we found that proline residue at the third N-terminal position (hereafter "P+3") promotes instability. Genetic perturbations identified the dipeptidyl peptidases DPP8 and DPP9 and the primary E3s of N-degron pathways, UBR proteins, as regulators of P+3 bearing substrate turnover. Interestingly, P+3 UBR substrates are significantly enriched for secretory proteins. We found that secretory proteins relying on a signal peptide (SP) for their targeting contain a "built-in" N-degron within their SP. This degron becomes exposed by DPP8/9 upon translocation failure to the designated compartments, thus enabling clearance of mislocalized proteins by UBRs to maintain proteostasis.

MeSH terms

  • Degrons
  • Dipeptidases / genetics
  • Dipeptidases / metabolism
  • Dipeptidyl-Peptidases and Tripeptidyl-Peptidases* / genetics
  • Dipeptidyl-Peptidases and Tripeptidyl-Peptidases* / metabolism
  • HEK293 Cells
  • Humans
  • Protein Sorting Signals
  • Protein Stability*
  • Proteolysis
  • Ubiquitin-Protein Ligases* / genetics
  • Ubiquitin-Protein Ligases* / metabolism

Substances

  • Dipeptidases
  • Dipeptidyl-Peptidases and Tripeptidyl-Peptidases
  • DPP8 protein, human
  • DPP9 protein, human
  • Protein Sorting Signals
  • Ubiquitin-Protein Ligases