Early and Late Effects of Low-Dose X-ray Exposure in Human Fibroblasts: DNA Repair Foci, Proliferation, Autophagy, and Senescence

Int J Mol Sci. 2024 Jul 28;25(15):8253. doi: 10.3390/ijms25158253.

Abstract

The effects of low-dose radiation exposure remain a controversial topic in radiation biology. This study compares early (0.5, 4, 24, 48, and 72 h) and late (5, 10, and 15 cell passages) post-irradiation changes in γH2AX, 53BP1, pATM, and p-p53 (Ser-15) foci, proliferation, autophagy, and senescence in primary fibroblasts exposed to 100 and 2000 mGy X-ray radiation. The results show that exposure to 100 mGy significantly increased γH2AX, 53BP1, and pATM foci only at 0.5 and 4 h post irradiation. There were no changes in p-p53 (Ser-15) foci, proliferation, autophagy, or senescence up to 15 passages post irradiation at the low dose.

Keywords: DNA damage response; DNA double-strand breaks; DNA repair foci; X-ray radiation; autophagy; fibroblasts; low-dose effects; proliferation; senescence.

MeSH terms

  • Autophagy* / radiation effects
  • Cell Proliferation* / radiation effects
  • Cells, Cultured
  • Cellular Senescence* / radiation effects
  • DNA Damage / radiation effects
  • DNA Repair* / radiation effects
  • Dose-Response Relationship, Radiation
  • Fibroblasts* / metabolism
  • Fibroblasts* / radiation effects
  • Histones / metabolism
  • Humans
  • Tumor Suppressor Protein p53 / genetics
  • Tumor Suppressor Protein p53 / metabolism
  • Tumor Suppressor p53-Binding Protein 1 / metabolism
  • X-Rays / adverse effects

Substances

  • Tumor Suppressor p53-Binding Protein 1
  • Histones
  • TP53BP1 protein, human
  • Tumor Suppressor Protein p53
  • H2AX protein, human