The likelihood of being helped or harmed as a patient-centred tool to assess ALK-Inhibitors clinical impact and safety in ALK-addicted non-small cell lung cancer: A systematic review and sensitivity-analysis

Cancer Treat Res Commun. 2024:41:100842. doi: 10.1016/j.ctarc.2024.100842. Epub 2024 Sep 6.

Abstract

Background: In untreated ALK-positive non-small cell lung cancer no randomized controlled trials (RCTs) are available directly comparing next-generation ALK-inhibitors. We conducted a sensitivity analysis using the likelihood of being helped or harmed (LHH).

Methods: Phase III trials comparing ALK-inhibitors to crizotinib were included. Efficacy outcomes were progression-free survival (PFS), objective response rate (ORR), PFS in patients with brain metastases and intracranial ORR. Safety outcomes were grade 3-4 adverse events (AEs), dose reductions and discontinuations.

Results: Six RCTs (1524 patients) were included. Lorlatinib and brigatinib had the lowest NNT for intracranial outcomes. Alectinib demonstrated favourable LHHs for grade 3-4 AEs, dose reductions and discontinuations. Brigatinib LHHs were low for common AEs, mainly laboratory anomalies and hypertension. Ensartinib showed mainly skin toxicity. Lorlatinib LHHs were low for specific grade 3-4 AEs, mainly metabolic alterations.

Conclusions: The four ALK-inhibitors exhibited favourable risk-benefit ratios. Lorlatinib showed the lowest NNT for systemic efficacy and, alongside with Brigatinib, lower NNTs for intracranial efficacy. Alectinib exhibited higher LHHs for AEs.

Registration: PROSPERO registration number: CRD42023389101.

Keywords: ALK-inhibitors; Alectinib; Brigatinib; Ensartinib; LHH; Lorlatinib.

Publication types

  • Systematic Review
  • Review

MeSH terms

  • Aminopyridines / adverse effects
  • Aminopyridines / pharmacology
  • Aminopyridines / therapeutic use
  • Anaplastic Lymphoma Kinase* / antagonists & inhibitors
  • Anaplastic Lymphoma Kinase* / genetics
  • Anaplastic Lymphoma Kinase* / metabolism
  • Carbazoles / pharmacology
  • Carbazoles / therapeutic use
  • Carcinoma, Non-Small-Cell Lung* / drug therapy
  • Carcinoma, Non-Small-Cell Lung* / pathology
  • Clinical Trials, Phase III as Topic
  • Crizotinib / pharmacology
  • Crizotinib / therapeutic use
  • Humans
  • Lactams / pharmacology
  • Lactams / therapeutic use
  • Lactams, Macrocyclic / pharmacology
  • Lactams, Macrocyclic / therapeutic use
  • Lung Neoplasms* / drug therapy
  • Lung Neoplasms* / pathology
  • Organophosphorus Compounds* / adverse effects
  • Organophosphorus Compounds* / pharmacology
  • Organophosphorus Compounds* / therapeutic use
  • Piperazines
  • Piperidines / adverse effects
  • Piperidines / pharmacology
  • Piperidines / therapeutic use
  • Progression-Free Survival
  • Protein Kinase Inhibitors* / adverse effects
  • Protein Kinase Inhibitors* / pharmacology
  • Protein Kinase Inhibitors* / therapeutic use
  • Pyrazoles / pharmacology
  • Pyrazoles / therapeutic use
  • Pyridazines
  • Pyrimidines / pharmacology
  • Pyrimidines / therapeutic use
  • Randomized Controlled Trials as Topic

Substances

  • Anaplastic Lymphoma Kinase
  • alectinib
  • Organophosphorus Compounds
  • brigatinib
  • lorlatinib
  • Protein Kinase Inhibitors
  • Carbazoles
  • Aminopyridines
  • ALK protein, human
  • Lactams
  • ensartinib
  • Piperidines
  • Pyrimidines
  • Crizotinib
  • Pyrazoles
  • Lactams, Macrocyclic
  • Piperazines
  • Pyridazines