Elucidation of the mechanism of the Yinhua Miyanling Tablet against urinary tract infection based on a combined strategy of network pharmacology, multi-omics and molecular biology

J Ethnopharmacol. 2025 Jan 30;337(Pt 1):118835. doi: 10.1016/j.jep.2024.118835. Epub 2024 Sep 16.

Abstract

Ethnopharmacological relevance: Yinhua Miyanling Tablet (YMT), a traditional Chinese medicine consisting of 10 herbs, has been widely used clinically to treat urinary tract infections (UTIs), however, its therapeutic mechanism is not fully understood.

Aim of the study: To investigate the mechanism of YMT in treating UTIs through network pharmacology, multi-omics and experimental validation.

Materials and methods: Clinically, blood and urine samples from YMT-treated UTI patients were collected for transcriptomic and metabolomic analyses. Computationally, compounds that are related to YMT were obtained from the databases, relevant targets were identified, and UTI-related targets were analyzed to determine the core signaling pathways. Subsequently, an integrated approach combining multi-omics and network pharmacology assisted in identifying the key pathways underlying therapeutic effects of YMT on UTI. Finally, a mouse model of UTI was established using uropathogenic Escherichia coli (UPEC), and the therapeutic mechanism of YMT on UTI was validated by ELISA, qRT-PCR and Western blotting.

Results: After taking YMT, patients showed reduced levels of urinary bacteria, white blood cells, and serum inflammatory factors (CRP, IL-6 and TNF-α). Multi-omics analysis combined with network pharmacology demonstrated that YMT significantly inhibited the TLR/MAPK/NFκB signaling pathway. In vivo experiments confirmed that YMT attenuated UPEC-induced pathological changes in bladder structural, reduced the expression of bladder proteins (TLR4, MyD88, p-p38 MAPK and p-p65 NFκB), increased protein expression of IκB-α, and attenuated the release of inflammatory factors (TNF-α, IL-6 and IL-1β) in mice.

Conclusion: YMT is effective in treating UTI by down-regulating the TLR4/p38MAPK/p65NFκB pathway, thereby providing a scientific basis for its clinical application.

Keywords: Metabolomics; Network pharmacology; Transcriptomics; Urinary tract infection; Yinhua Miyanling Tablet.

MeSH terms

  • Adult
  • Animals
  • Disease Models, Animal
  • Drugs, Chinese Herbal* / pharmacology
  • Escherichia coli Infections / drug therapy
  • Female
  • Humans
  • Male
  • Metabolomics
  • Mice
  • Middle Aged
  • Multiomics
  • Network Pharmacology*
  • Signal Transduction / drug effects
  • Tablets*
  • Urinary Tract Infections* / drug therapy
  • Urinary Tract Infections* / microbiology
  • Uropathogenic Escherichia coli / drug effects

Substances

  • Drugs, Chinese Herbal
  • Tablets