The MdERF61-mdm-miR397b-MdLAC7b module regulates apple resistance to Fusarium solani via lignin biosynthesis

Plant Physiol. 2024 Dec 23;197(1):kiae518. doi: 10.1093/plphys/kiae518.

Abstract

Apple replant disease (ARD) is a worldwide problem that threatens the industry. However, the genetic mechanism underlying plant disease resistance against ARD remains unclear. In this study, a negative regulatory microRNA in Malus domestica, mdm-miR397b, and its direct target MdLAC7b (Laccase) was selected for examination based on our previous small RNA and degradome sequencing results. Overexpressing the mdm-miR397b-MdLAC7b module altered the lignin deposition and jasmonic acid contents in apple roots, which also led to increased resistance to Fusarium solani. Additionally, Y1H library screening using mdm-miR397b promoter recombinants identified a transcription factor, MdERF61, that represses mdm-miR397b transcriptional activity by directly binding to 2 GCC-boxes in the mdm-miR397b promoter. In summary, our results suggest that the MdERF61-mdm-miR397b-MdLAC7b module plays a crucial role in apple resistance to F. solani and offers insights for enhancing plant resistance to soil-borne diseases in apples.

MeSH terms

  • Disease Resistance* / genetics
  • Fusarium* / pathogenicity
  • Fusarium* / physiology
  • Gene Expression Regulation, Plant*
  • Lignin* / biosynthesis
  • Lignin* / metabolism
  • Malus* / genetics
  • Malus* / immunology
  • Malus* / microbiology
  • MicroRNAs* / genetics
  • Plant Diseases* / genetics
  • Plant Diseases* / immunology
  • Plant Diseases* / microbiology
  • Plant Proteins* / genetics
  • Plant Proteins* / metabolism

Substances

  • Lignin
  • MicroRNAs
  • Plant Proteins

Supplementary concepts

  • Fusarium solani