Abstract
Here, RNAi library screening reveals that multiple G2/M and kinetochore components, including TTK/monopolar spindle 1, modulate TNFα-induced NF-κB activation, cell survival, and genotoxicity, underscoring their potential importance as therapeutic targets in HNSCC.
©2024 The Authors; Published by the American Association for Cancer Research.
MeSH terms
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Carcinoma, Squamous Cell* / genetics
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Carcinoma, Squamous Cell* / metabolism
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Carcinoma, Squamous Cell* / pathology
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Cell Line, Tumor
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Cell Survival / drug effects
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Head and Neck Neoplasms* / genetics
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Head and Neck Neoplasms* / metabolism
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Head and Neck Neoplasms* / pathology
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Humans
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Kinetochores* / metabolism
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NF-kappa B* / metabolism
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RNA Interference*
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Signal Transduction*
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Squamous Cell Carcinoma of Head and Neck / genetics
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Squamous Cell Carcinoma of Head and Neck / metabolism
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Squamous Cell Carcinoma of Head and Neck / pathology
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Tumor Necrosis Factor-alpha* / genetics
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Tumor Necrosis Factor-alpha* / metabolism
Substances
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NF-kappa B
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Tumor Necrosis Factor-alpha